近期论文
nayak g, cooper gm. (2012). p53 is a major component of the transcriptional and apoptotic program regulated by pi 3-kinase/akt/gsk3 signaling. cell death dis. 3:e400.
tullai jw, tacheva s, owens lj, graham jr, cooper gm. (2011). ap-1 is a component of the transcriptional network regulated by gsk-3 in quiescent cells. plos one 6: e20150.
terragni j, nayak, g, banerjee s, medrano jl, graham jr, brennan jf, sepulveda s, cooper gm. (2011). the e-box binding factors max/mnt, mitf and usf1 act coordinately with foxo to regulate expression of pro-apoptotic and cell cycle control genes by phosphatidylinositol 3-kinase/akt/gsk3 signaling. j. biol. chem. 286: 36215-36227.
mullenbrock s, shah j, cooper gm. (2011). global expression analysis identified a preferentially nerve growth factor-induced transcriptional program regulated by sustained mitogen-activated protein kinase/extracellular signal-regulated kinase (erk) and ap-1 activation during pc12 differentiation. j. biol. chem. 286:45131-45145.
graham, j.r., hendershott, m.c., terragni, j. and cooper, g.m. (2010). mrna degradation plays a significant role in the program of gene expression regulated by phosphatidylinositol 3-kinase signaling. mol. cell. biol. 30: 5295-5305.
graham jr, tullai jr and cooper gm. (2010). gsk-3 represses growth factor-inducible genes by inhibiting nf-kb in quiescent cells. j. biol. chem. 285: 4472-4480.
terragni j, graham, jr, adams kw, schaffer mw, tullai jw, cooper gm. (2008). phosphatidylinositol 3-kinase signaling in proliferating cells maintains an anti-apoptotic transcriptional program mediated by inhibition of foxo and non-canonical activation of nfkb transcription factors. bmc cell biol. 9, 6.
tullai jw, schaffer me, mullenbrock s, sholder g, kasif s, cooper gm. (2007). immediate-early and delayed primary response genes are distinct in function and genomic architecture. j. biol. chem. 282, 23981-23995.
